促黃體生成激素（兔多克隆抗體）

LH 促黃體生成激素（兔多克隆抗體）

廣州健侖生物科技有限公司

促黃體生成素是垂體細(xì)胞分泌的一種激素。此抗體和人的促黃體生成素反應(yīng)，和促甲狀腺素、卵泡刺激素或絨毛膜促性腺激素有微弱交叉反應(yīng)，主要用于垂體腺瘤功能性分類的研究。

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

歡迎咨詢

歡迎咨詢

LH 促黃體生成激素（兔多克隆抗體）

【產(chǎn)品介紹】

細(xì)胞定位：細(xì)胞漿

適用組織：石蠟/冰凍

陽(yáng)性對(duì)照：垂體

抗原修復(fù)：熱修復(fù)（檸檬酸）

抗體孵育時(shí)間：30-60min

LH

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】     歐

【】 
【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

從花栗鼠和松鼠的角度來(lái)思考這一問(wèn)題：減少生物系統(tǒng)中的花栗鼠種群或許可使得松鼠興旺——尤其如果是減少花栗鼠的途經(jīng)改變了生態(tài)系統(tǒng)從而有利于松鼠，這與輻射改變機(jī)體來(lái)支持C/EBPα 突變干細(xì)胞相類似。
這些研究不只是告訴了我們?yōu)槭裁摧椛淇墒乖煅杉?xì)胞分化;它們還證實(shí)了通過(guò)激活一條干細(xì)胞維持信號(hào)通路，我們可以阻止其發(fā)生。甚至在輻射后數(shù)月，人為地激活受輻射HSCs的NOTCH信號(hào)通路可以讓它們?cè)俣缺憩F(xiàn)“干性”——在這些本該響應(yīng)輻射發(fā)生分化的HSCs中重啟血細(xì)胞組裝。
當(dāng)DeGregori、Fleenor和同事們?cè)谝酝馐茌椛涞腍SCs中激活NOTCH時(shí)，遏制住了危險(xiǎn)的C/EBPα 細(xì)胞群。與通過(guò)激活NOTCH恢復(fù)適應(yīng)性的非C/EBPα 突變干細(xì)胞競(jìng)爭(zhēng)，C/EBPα 突變干細(xì)胞沒(méi)有了進(jìn)化的空間。
DeGregori說(shuō)：“如果我在一種有可能經(jīng)受全身輻射的環(huán)境中工作，我會(huì)冷凍一堆我的HSCs。在輻射照射后輸注一次健康HSCs將有可能使得健康血液系統(tǒng)在競(jìng)爭(zhēng)中勝過(guò)‘具有編程能力’的輻射照射HSC，甚至是攜帶致癌突變的HSC。希望在未來(lái)，我們能夠提供一些藥物來(lái)恢復(fù)輻射后留下的干細(xì)胞的適應(yīng)性”。

Consider this from the chipmunk and squirrel perspectives: Reducing the population of chipmunk in the biological system may allow squirrels to thrive - especially if the path to the chipmunk is diminished by altering the ecosystem to benefit the squirrel, To support similar C / EBPα mutant stem cells.
These studies are not just ling us why radiation causes hematopoietic stem cell differentiation; they also confirm that by activating a stem cell to maintain the signaling pathway, we can stop it from happening. Even several months after irradiation, the NOTCH signaling pathway that artificially activates irradiated HSCs can render them "dry" again - restarting blood cell assembly in these HSCs that would otherwise respond to radiation differentiation.
When DeGregori, Fleenor and colleagues activated NOTCH in irradiated HSCs, they blocked the dangerous C / EBPα cell population. In competition with non-C / EBPα mutant stem cells that activate adaptive NOTCH recovery, C / EBPα mutant stem cells have no room for evolution.
"If I work in an environment that has the potential to experience whole-body radiation, I will freeze a bunch of my HSCs," said DeGregori. "Infusing a healthy HSC after radiation exposure will likely make the healthy blood system outbid ' Programmed ability 'to irradiate HSCs, even HSCs carrying oncogenic mutations, and hopefully in the future we will be able to offer some drugs to restore the resilience of stem cells left after radiation. "