衣原體Chlamydia酶聯(lián)免疫法ELISA 檢測試劑

衣原體Chlamydia酶聯(lián)免疫法ELISA 檢測試劑

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沙眼衣原體診斷試劑盒（ELISA法)

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衣原體Chlamydia酶聯(lián)免疫法ELISA 檢測試劑

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-3室
【企業(yè)文化】

The duration of exposure to water is proportional to the number of times. Sometimes it is infested by the cercariae when it is swallowed or when it is swallowed. (3) Susceptibility Humans and vertebrates are generally susceptible to schistosomiasis. The prevalence of infection among school-age children and adolescents is highest in endemic areas and gradually declines afterwards, which is related to protective immunity. Disease-associated editing Pathogenesis Schistosomiasis cercariae, schistosomiasis and eggs produce mechanical damage to the host and cause complex immune-pathological responses. When the cercaria penetrates the skin, it causes dermatitis. The dermatitis occurs only in people who have infected the cercariae. It is an immediate and delayed type reaction. Caudal dermatitis has a certain role in promoting the breakage of schistosomula in the skin, and is the response of the host's acquired immunity to reinfection. When the larvae migrate in the body, they mainly pass through the lungs, causing vasculitis, capillary embolism, rupture, and local cell infiltration and spotting. Patients may present with cough, hemoptysis, fever, eosinophilia, etc. The damage caused by the migration of schistosomiasis is related to the reaction caused by the parasite metabolites. Adult metabolites can form immune complexes, causing systemic reactions and local vascular lesions and tissue lesions; living in the portal vein system, can cause mild endocarditis and perivenitis; dead insects can flow into the liver with blood, in the embolism Causes inflammation of the surrounding tissue. Schistosomiasis infection can lead to a decrease in overall immune function, which exacerbates the development of concomitant diseases or concurrent infections. Infiltration of cells around the eggs forms egg granuloma. The formation and development of granuloma are closely related to the degree of development of the eggs. When the eggs have not yet formed trichomes, the surrounding tissues may have only mild reactions. After the pods mature in the eggs, granuloma reactions are caused by soluble antigens such as enzymes, proteins, and sugars secreted by the eggs. The development of granuloma is consistent with the development process of the eggs, and the local exudation and hyperplasia reactions gradually increase. After calcification of the eggs, the granuloma begins to degenerate to form fibrous scar tissue. This reaction not only helps to destroy and eliminate eggs, but also makes the eggs antigens localized, reducing the impact on the whole body. Granuloma can affect the host's liver and intestinal tissue, resulting in cirrhosis and fibrosis of the intestinal wall. At present, it is believed that under the stimulation of eggs soluble antigens, the host produces corresponding antibodies, antigens and antibodies form complexes around the eggs, causing local reactions, which is the main mechanism of granuloma formation by Schistosoma japonicum. On the other hand, the granulomatous reaction contributes to the destruction and elimination of the eggs, and confines the oozing of the eggs to the eggs, in order to reduce and avoid systemic damage caused by antigen-antibody complexes. With the development of the infection process, the intensity of granulomatous reaction is gradually weakened. As the host's immune regulation, the ability to destroy the eggs continues to increase, playing a role in protecting the host. Humans have no innate immunity to schistosome and may have protective immunity.